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Model for the control of Noxa and Puma expression during DNA... | Download Scientific Diagram
Clues from worms: a Slug at Puma promotes the survival of blood progenitors | Cell Death & Differentiation
Schematic diagram of p53 and PUMA regulation of apoptosis following... | Download Scientific Diagram
A p53/lnc‐Ip53 Negative Feedback Loop Regulates Tumor Growth and Chemoresistance - Zhang - 2020 - Advanced Science - Wiley Online Library
Therapeutic Response to Non-genotoxic Activation of p53 by Nutlin3a Is Driven by PUMA-Mediated Apoptosis in Lymphoma Cells - ScienceDirect
Both p53–PUMA/NOXA–Bax-mitochondrion and p53–p21cip1 pathways are involved in the CDglyTK-mediated tumor cell suppression - ScienceDirect
Regulation of transactivation-independent proapoptotic activity of p53 by FOXO3a
6-OHDA generated ROS induces DNA damage and p53- and PUMA-dependent cell death | Molecular Neurodegeneration | Full Text
PUMA Couples the Nuclear and Cytoplasmic Proapoptotic Function of p53 | Science
A review of the role of Puma, Noxa and Bim in the tumorigenesis, therapy and drug resistance of chronic lymphocytic leukemia | Cancer Gene Therapy
Slugging It out: Fine Tuning the p53-PUMA Death Connection: Cell
The role of P53 up-regulated modulator of apoptosis (PUMA) in ovarian development, cardiovascular and neurodegenerative diseases | SpringerLink
p53 and PUMA: A Deadly Duo | Science
PDF] Importance of proapoptotic protein PUMA in cell radioresistance. | Semantic Scholar
Puma, noxa, p53, and p63 differentially mediate stress pathway induced apoptosis | Cell Death & Disease
BH3-only sensors Bad, Noxa and Puma are Key Regulators of Tacaribe virus-induced Apoptosis | PLOS Pathogens
The p53-Target Gene Puma Drives Neutrophil-Mediated Protection against Lethal Bacterial Sepsis | PLOS Pathogens
The nuclear function of p53 is required for PUMA-mediated apoptosis induced by DNA damage | PNAS
PUMA decreases the growth of prostate cancer PC-3 cells independent of p53
The Role of p53 in Apoptosis - Joana D Amaral - Discovery Medicine
p53: More than a Transcription Factor?: R&D Systems
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